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Recognition of tirzepatide The tirzepatide–GIPR–G s and tirzepatide–GLP-1R–G s exhibit a similar peptide-receptor binding interface, where distinct structural features were observed at ECL1, ECL3 and the extracellular tips of TM1 and TM3 (Fig
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Semaglutide reduced systolic blood pressure by 3.3 mm Hg, high-sensitivity C-reactive protein by 37.8%, and improved lipid profiles—all achieved on top of high baseline rates of evidence-based cardiovascular therapies (90% on statins, 86% on antiplatelet agents).[1] These pleiotropic effects likely contribute to cardiovascular risk reduction through mechanisms distinct from adiposity changes, supporting the reconceptualization of GLP-1 receptor agonists as cardiovascular disease-modifying agents rather than simply weight-loss medications
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